Metabolism can roughly be defined as the chemistry that turns food into life. Insulin and leptin, therefore, are critical to health and disease. Insulin and leptin work together to control the quality of metabolism (and, to a significant extent, the rate of metabolism).
Insulin works mostly at the individual cell level, telling the vast majority of cells whether to burn or store fat or sugar and whether to utilize that energy for maintenance and repair or reproduction. This is extremely important, because, at the individual cell level, turning on maintenance and repair equates to increased longevity. Turning up cellular reproduction, on the other hand, increases the risk of cancer.
Genetic studies in simple organisms have convincingly shown the link between energy stores, reproduction and aging to be at least partially mediated by insulin (which in simple organisms also functions as growth hormone) When insulin signals are kept low, indicating scarce energy availability, whether or not, in fact, energy is scarce, lifespan can be greatly extended.
Levels of insulin are largely determined by glucose (and amino acid from protein) levels. In many people with diabetes, insulin levels are also determined by how much insulin they are taking. Many have been told that what they eat does not matter as long as they take enough insulin to cover it.
This couldn't be further from the truth. Consider two people with equivalent blood sugar levels but who are taking different amounts of insulin. The person taking higher amounts of insulin will likely age faster and accumulate the "diseases" associated with aging, such as heart disease, obesity, and even diabetes itself. Yes, that person's diabetes will get worse. Why? Because most cases of type 2 diabetes are caused by overexposure to insulin.
Just as you become unable to smell the odor in a smelly room after having been there awhile, your cells become unable to "smell" the essential messages from insulin (and leptin) after they have been exposed to high levels of these hormones. Your body responds to this inadequate signalling either by producing higher levels of insulin and leptin or by requiring more to get the message across, contributing to a vicious cycle.
In short, low insulin is very healthy and good for you as long as its message is being heard. Most treatments for type 2 insulin-resistant diabetes, however, involve drugs that raise insulin or utilize injections of insulin itself. Treatment of type I diabetes also generally requires excessive quantities of insulin.
The tragic result is that conventional medical treatment for diabetes contributes to the manifest side effects and the shortened lifespan that people with diabetes experience. The major cause of poor results from conventional diabetes treatments is not that people with diabetes are inadequate students who simply need more education. The major problem is actually what is being taught, and this needs to be changed.
If you have lost the ability to smell an odor in a room, the best way to smell it is not to make the smell stronger (analogous to taking or making more insulin). What you should do is walk out of the room to re-sensitize your nose. When you walk back in, you can smell the odor well again, even if it has been reduced.
This is how diabetes needs to be treated. We need to reduce, not increase, the levels of insulin, so that your cells can "smell" it better. You need to reduce not just your blood suger , but also your insulin, and the only way to do that is to get "more bang out of each insulin buck" by increasing insulin sensitivity. Contrary to what you have been told, there are no insulin sensitizing drugs. Only changing what you eat (combined perhaps with supplements that augment the dietary changes) can do this. Fortunately, changing diet does this very well, well enough to reverse most cases of type 2 diabetes completely.
Source :Dr. Ron Rosedale
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